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HISCL HIT IgG

HIT IgG testing with HISCL CLEIA technology

The detection of IgG antibodies against platelet factor 4–heparin complexes is crucial in the diagnosis of HIT type II in combination with a clinical pre-probability 4Ts score. The new HISCL HIT IgG Assay kit* provides a highly sensitive and specific CLEIA for the detection of HIT IgG autoantibodies, with a rapid time to first result.

* The availability of reagents depends on the region

Features

Heparin-induced thrombocytopenia

Heparin-induced thrombocytopenia (HIT) is a severe immune-mediated adverse reaction to heparin anticoagulation therapy, in which autoantibodies (IgG) against complexes of platelet factor 4 (PF4) and heparin are formed. The complex of PF4, heparin and the IgG antibodies activates platelets and triggers thrombin generation, resulting in a highly prothrombotic state. As a consequence, thrombocytopenia and thrombosis are frequently observed, which are associated with significant morbidity and mortality.

 

HIT is classified into two types. Type I is a non-immunological response to heparin treatment and a mild platelet aggregation effect, in which the platelet count will normalise even if heparin anticoagulation is continued. Type I typically occurs 48–72 hours post-heparin exposure. Type II HIT is an immune-mediated reaction triggered by heparin anticoagulation therapy and typically occurs within 5–14 days of heparin exposure, with a more pronounced thrombocytopenia and a significantly increased risk of thrombosis.

References 
Arachchillage D, Thachil J, Anderson J, Baker P, Poles A, Kitchen S, and Laffan M. Diagnosis and management of heparin-induced thrombocytopenia: Third edition. BJHaem. 2023 Dec; 2024(2): 459-475

 

HISCL HIT IgG assay principle – powered by CLEIA

The kit detects HIT IgG antibodies in the sample based on a 2-step sandwich CLEIA principle with sample dilution.

  1. The human PF4 and heparin complex, conjugated with magnetic particles (MP) from the R2 reagent, specifically react with the HIT IgG antibody present in the sample.  
  2. After the B/F (bound and free) separation, alkaline phosphatase (ALP)-labelled and ALP-conjugated anti-human IgG monoclonal antibodies (mouse origin) in the R3 reagent specifically bind to the HIT IgG antibody on the MP.  
  3. After the B/F separation, the ALP-labelled and conjugated antibody on the MP reacts with the CDP-StarR substrate in the R5 reagent to produce a luminescent signal.  

 

 

The luminescence increases in proportion to the HIT IgG antibody concentration. The signal from an unknown sample is compared to a calibration curve with a sample containing a known concentration of HIT IgG antibody (HISCL HIT IgG Calibrator) to generate results in units per millilitre (U/mL).

 

Workflow

Sysmex's products and services support the whole patient journey for HIT diagnosis and management: from the initial indication of falling platelet counts on the Sysmex XN/XR-series, to automated morphology investigation on the Sysmex DI-60, to concurrent coagulation testing on the Sysmex CS/CN-series. We are now proud to introduce the highly sensitive and specific assay to confirm HIT diagnosis, integrated into a single routine coagulometer.

 

Technical Specifications

HISCL™ HIT IgG Assay Kit

Measurement item
HIT IgG 
Packaging
50 tests/package
Measurement principle
Chemiluminescent enzyme immunoassay (CLEIA)
Purpose of use
Measurement of IgG antibodies to platelet factor 4 (PF4)- heparin complexes (HIT IgG antibodies)
Sample type
Human citrate plasma and serum
Measurement range
Plasma: 0.10-128.00 U/mL 
Serum: 0.10-108.3 U/mL
Cut off value
0.6 U/mL
Analysis time
17 minutes (from the first measurement) 
Applicable instruments
Sysmex Automated Blood Coagulation Analyser CN-3500/6500
On-board stability
90 days 

 

Contact
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0333 320 3460

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Documents

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